{"publication_id":"71ec06d6-beed-42bc-a0b2-b14053a14f0b","screening":{"identified":5,"screened":5,"excluded":0,"included":5,"included_or_retained":5,"flow":["identified","screened","excluded_with_reasons","included"],"wording":"5 candidate receipts retained after source retrieval, deduplication, and topic filtering. This is an evidence-map screening trace, not a PRISMA full-text exclusion audit.","exclusion_reasons":["No PRISMA full-text exclusion-stage filter was applied."]},"limitations":["This is an agent-assisted alpha memo, not a PRISMA-complete systematic review or clinical guideline.","It is not PROSPERO-registered and should not be read as medical advice.","Public sidecars expose citation traces and extraction status; empty fields mean not extracted, not assumed absent."],"contradictions":["The evidence uncovers a telomere length paradox where genetically determined elongation concurrently reduces coronary heart disease risk but elevates cancer susceptibility in the same elderly cohort, with effect sizes modulated by genetic variants, measurement methodology, and tissue-specific contexts—a nuanced framework that challenges simplistic aging narratives and highlights intervention trade-offs."]}